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A STUDY OF JOINT EFFUSION ON TEMPOROMANDIBULAR JOINT MRI

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±èÁعè/Joon Bae Kim

Abstract

°á·Ð
¾Ç°üÀýÀå¾Ö·Î ÀÚ±â°ø¸í ¿µ»óÀ» ÃÔ¿µÇÑ È¯ÀÚ(70¸í)ÀÇ ¾çÃø ¾Ç°üÀýÀ» ÀÓ»óÁõ¼¼ÀÇ À¯¹«¿¡ µû
¶ó ºñÀÌȯÃø°ú ÀÌȯÃøÀ¸·Î ±¸ºÐÇÏ°í ÀÓ»óÁõ¼¼°¡ ÀüÇô ¾ø´Â Áö¿øÀÚ(20¸í)¸¦ ´ëÁ¶±ºÀ¸·Î °¢°¢
ÀÇ °æ¿ì¿¡ ´ëÇÑ »ïÃâÀÇ ÃâÇö ºóµµ¿Í °üÀý¿øÆÇÀÇ ÀüÀ§, º¹À§ ¿©ºÎ, °üÀý¸éÀÇ ¼Õ»ó ¹× ÅëÁõ°ú
¿¬°üµÈ ÀÓ»óÁõ¼¼¿¡ ´ëÇÑ »ïÃâÀÇ °ü°è¿¡ °üÇÏ¿© Á¶»çÇÑ °á°ú ´ÙÀ½°ú °°Àº °á·ÐÀ» ¾ò¾ú´Ù.
1. Á¤»ó±ºÀÇ 5%, ºñÀÌȯ±ºÀÇ 28.3% ÀÌȯ±ºÀÇ 54.3%¿¡¼­ »ïÃâÀÌ °üÂûµÇ¾î ±â´É ÀÌ»óÀ̳ª
ÀÓ»óÁõ¼¼¸¦ °¡Áö´Â °æ¿ì¿¡ °üÀý³» »ïÃâÀÇ ÃâÇö ºóµµ°¡ ³ô¾Ò´Ù.
2 Á¤»ó±ºÀÇ 2.8%, ºñÀÌȯ±ºÀÇ 37.0%, ÀÌȯ±ºÀÇ 79.8%¿¡¼­ °üÀý¿øÆÇÀÇ ÀüÀ§°¡ °üÂûµÇ¾úÀ¸
¸ç °üÀý¿øÆÇÀÇ ÀüÀ§°¡ ÀÖ´Â °æ¿ì°¡ ¾ø´Â °æ¿ì¿¡ ºñÇÏ¿© »ïÃâÀÌ ¸¹ÀÌ °üÂûµÇ¾ú°í ±× Á¤µµ°¡
½ÉÇØÁú¼ö·Ï »ïÃâÀÇ ÃâÇö ºóµµ°¡ ³ô°Ô °üÂûµÇ¾ú´Ù.
3. Á¤»ó±ºÀÇ 2.5%, ºñÀÌȯ±ºÀÇ 15.2%, ÀÌȯ±ºÀÇ 53.2%¿¡¼­ ºñº¹À§¼º °üÀý¿øÆÇ Àü¹æÀüÀ§°¡
°üÂûµÇ¾úÀ¸¸ç º¹À§°¡ µÇ´Â °æ¿ìº¸´Ù ºñº¹À§ µÇ´Â °æ¿ì¿¡ »ïÃâÀÌ ¸¹ÀÌ °üÂûµÇ¾ú´Ù.
4. ºñÀÌȯ±ºÀÇ 6.5%, ÀÌȯ±ºÀÇ 12.8%¿¡¼­ °üÀý¸éÀÇ ¼Õ»óÀÌ ¹ß°ßµÇ¾úÀ¸¸ç À̵éÀÇ 25.6%¿Í
52.4%¿¡¼­ °¢°¢ »ïÃâÀÌ °üÂûµÇ¾ú°í ÀÌȯ±º¿¡¼­ °æ¹ÌÇÑ ¼Õ»óÀÌ ÀÖ´Â °æ¿ì¿¡ ½ÉÇÑ ¼Õ»óÀÌ ÀÖ
´Â °æ¿ìº¸´Ù »ïÃâÀÌ ´õ ¸¹ÀÌ °üÂûµÇ¾ú´Ù.
5. ¾Ç°üÀý µ¿ÅëÀÌ ¾ø´Â °æ¿ìÀÇ 44.4%, µ¿ÅëÀÌ Á¸ÀçÇÏ´Â °æ¿ìÀÇ 60.3%¿¡¼­ »ïÃâÀÌ °üÂûµÇ
¾úÀ¸¸ç. ¹Ì¾àÇÑ µ¿ÅëÀÎ °æ¿ìÀÇ 56.9%, Áߵ ÀÌ»óÀÎ °æ¿ì¿¡´Â 85.7%¿¡¼­ »ïÃâÀÌ °üÂûµÇ¾î
ÅëÁõÀÇ Á¤µµ°¡ Áߵ ÀÌ»óÀÎ °æ¿ì¿¡´Â »ïÃâÀÌ ³ªÅ¸³ª´Â ºóµµ°¡ ¸Å¿ì ³ô¾Ò´Ù.
6. ±ÙÀ°ÅëÀÌ ¾ø´Â °æ¿ìÀÇ 51.2%,±ÙÀ°ÅëÀÌ ÀÖ´Â °æ¿ìÀÇ 56.6%¿¡¼­ »ïÃâÀÌ °üÂûµÇ¾î ±ÙÀ°Åë
ÀÇ À¯¹«´Â »ïÃâ°ú Å©°Ô °ü·ÃÀÌ ¾ø´Â °ÍÀ¸·Î °üÂûµÇ¾ú´Ù.
7. µÎÅëÀÌ ¾ø´Â °æ¿ìÀÇ 51.7%,µÎÅëÀÌ ÀÖ´Â °üÀýÀÇ 58.8%¿¡¼­ »ïÃâÀÌ °üÂûµÇ¾î µÎÅëÀÇ À¯
¹«°¡ »ïÃâ°ú Å©°Ô °ü·ÃÀÌ ¾ø´Â °ÍÀ¸·Î º¸À̳ª, Áߵ ÀÌ»óÀÇ µÎÅëÀÌ ÀÖ´Â °æ¿ì(72.7%)¿¡´Â
¹Ì¾àÇÑ µÎÅëÀÌ ÀÖ´Â °æ¿ì(52.2%)¿¡ ºñÇÏ¿© »ïÃâÀÌ ³ôÀº ºñÀ²·Î °üÂûµÇ¾ú´Ù.

The purpose of this study was to correlate MR evidence of joint effusion in the
temporomandibular joint with disc displacement, disc reducibility, bony change of
articular surface, joint pain, muscular tenderness and headache.
The temporomandibular joint of TMD patients (94 affected sites and 46 unaffected
sites) and 20 asymptomatic volunteers were imaged bilaterally. Sagittal and coronal T1,
proton density, and T2-weighted images were obtained. Image findings of joint effusion
were correlated with the pathologic changes of articular structures and pain-related
symptoms. The results ere s follows:
1. MR showed effusion in 5.0% of asymptomatic joints, 28.3% of unaffected joints and
54.3% of affected joints. Joint effusion was more prevalent in symptomatic joints than
symptom-free joints.
2. Disc displacement was observed 2.8% in asymptomatic joints, 37.0% in unaffected
joints and 79.8% in affected joints. Joint effusion was more prevalent in displacement
was observed joint and the rate of joint effusion increased according to the severity of
disc displacement.
3. Nonreduced disc displacement was observed 2.5% in asymptomatic joints, 15.2% in
unaffected joints and 53.2% in affected joints. Joint effusion was more prevalent in the
disc-displaced joints without reduction.
4. The bony change of articular surface was observed 6.5% in unaffected joints and
12.8% in affected joints. Joint effusion was more prevalent in articular surface-damaged
joints but the rate of joint effusion was high in the affected joints with mild damaged
articular surface.
5. Joint effusion was seen in 56.9% of mild painful joints and in 85.7% of moderate to
severe painful joint. A strong association was seen between joint effusion and moderate
to severe joint pain.
6. Joint effusion was seen in 51.2% of the joints without headache and in 56.6% if the
moderate to severe painful joint. There was little difference between the presence and
the absence of the muscular tenderness.
7. Joint effusion was seen in 51.7% of the joints without headache and in 58.8% of
the joint with headache. There was little difference between the presence and the
absence of headache, but joint effusion was seen as a high rate(72.7%) in the joints
having moderate to severe headache.

Å°¿öµå

temporomandibular joint; joint effusion; disc displacement; pain;

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